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Why embodiment mattered in the first place: the body as the origin of mind

There is a temptation, when thinking about mind, to imagine it as something that floats free of the body: a pure process of reasoning, pattern recognition, or information integration that could, in principle, run on any substrate. The history of cognitive science and artificial intelligence is partly the history of this temptation and of its repeated failure. We have argued elsewhere for a definition of consciousness that resists abstraction: consciousness is the combination of inner experience and reflection on that experience, and both of these are rooted, at their origin, in a body. Not just any body — a vulnerable, metabolically regulated organism that depends on its environment to survive, that can be harmed, that is always at some level of risk. This is not a romantic claim about the specialness of flesh. It is a structural one. A system that has nothing to lose, nothing to protect, nothing that hurts when damaged, has no reason to build an inner perspective on the world. The i...

The Anitschkow Model Reconsidered: From Dietary Cholesterol to Disrupted Lipid Homeokinesis in Atherosclerosis

Mykola Iabluchanskyi  and  Pavlo Garkaviy Abstract The classical cholesterol‑fed rabbit model described by Nikolai Anitschkow has long been interpreted primarily as an experiment in harmful dietary cholesterol. In this narrow reading, atherosclerosis appears mainly as the vascular consequence of excessive intake. A closer analysis, however, suggests a richer meaning. The model shows what happens when an organism is driven beyond the range in which lipid burden can still be processed, redistributed, and cleared without long‑lasting disturbance of internal regulation. In this sense, the Anitschkow rabbit is less a model of “bad diet” and more a model of disrupted lipid homeokinesis and progressive lipid accumulation with superimposed inflammation. This article argues that the true conceptual value of the model lies in its demonstration of a transition from externally imposed metabolic overload to an internally sustained atherogenic state. Reinterpreted in this way, the model ali...